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                <identifier>oai:data.mendeley.com/f3w23wkxw7.1</identifier>
                <datestamp>2017-05-28T02:59:59Z</datestamp>
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            <metadata><oai_dc:dc xsi:schemaLocation="http://www.openarchives.org/OAI/2.0/oai_dc/ http://www.openarchives.org/OAI/2.0/oai_dc.xsd" xmlns:oai_dc="http://www.openarchives.org/OAI/2.0/oai_dc/" xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
    <dc:creator>Liu, Yang</dc:creator>
    <dc:title>miR-371b-5p and miR-155-5p</dc:title>
    <dc:publisher>Mendeley Data</dc:publisher>
    <dc:description>These findings in accordance with our results that miRNAs involved in the pathogenesis of metabolic disorders in EAT of CAD patients. 
Overall, we were able to depict a novel miRNA signature of EAT in CAD patients characterized by a disregulated of miRNAs profile that are probably involved in pathogenesis of CAD. 
The results were validated by qRT-PCR and cell transfection. 
The results of the current study support the hypothesis that miRNA expression is deregulated in epicardial adipose tissue in patients suffering from CAD disease.

We provided a comprehensive potential novel miRNAs expression signature of EAT in CAD patients, and we showed that the targets of the miRNAs are necessarily associated to the pathogenesis of CAD.</dc:description>
    <dc:subject>Bioinformatics</dc:subject>
    <dc:subject>Microarray</dc:subject>
    <dc:subject>RNA Gene</dc:subject>
    <dc:type>Dataset</dc:type>
    <dc:identifier>doi:10.17632/f3w23wkxw7.1</dc:identifier>
    <dc:identifier>oai:data.mendeley.com/f3w23wkxw7.1</dc:identifier>
    <dc:rights>Creative Commons Attribution 4.0 International</dc:rights>
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    <dc:date>2017-05-28T02:59:59Z</dc:date>
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