Hyperactive end joining repair mediates resistance to DNA damaging therapy in p53-deficient cells

Published: 5 December 2020| Version 1 | DOI: 10.17632/hndkgw35jb.1
Contributors:
Rashmi Kumar,
Dennis Simpson,
Gaorav Gupta

Description

Single-cell tracking and foci counting of si-Control vs. si-TP53 treated RPE1 cells. Cells are also divided by exposure to DNA Damaging Agents (NCS +/- DNA-PKi).

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Steps to reproduce

Please refer to manuscript DOI: 10.17632/hndkgw35jb.1 for methods on analysis of live-cell imaging datasets.