Hyperactive end joining repair mediates resistance to DNA damaging therapy in p53-deficient cells
Single-cell tracking and foci counting of si-Control vs. si-TP53 treated RPE1 cells. Cells are also divided by exposure to DNA Damaging Agents (NCS +/- DNA-PKi).
Steps to reproduce
Please refer to manuscript DOI: 10.17632/hndkgw35jb.1 for methods on analysis of live-cell imaging datasets.