Effects of differential external pneumatic compression pressures on recovery and repeated sprint performance: a randomized placebo-controlled crossover study

Published: 24 August 2026| Version 1 | DOI: 10.17632/jcthkyfsvd.1
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This dataset contains the anonymized raw data supporting the study entitled "Effects of differential external pneumatic compression pressures on recovery and repeated sprint performance: a randomized placebo-controlled crossover study." Twelve recreationally trained men completed four experimental conditions in a randomized, single-blind, placebo-controlled crossover design: passive control (CON), placebo compression (PLA), external pneumatic compression at 90 mmHg, and external pneumatic compression at 190 mmHg. Each trial included a repeated-sprint cycling fatigue protocol, a 20-min condition-specific recovery intervention, a subsequent repeated-sprint performance test consisting of five 10-s maximal sprints separated by 60 s of passive recovery, and a final 30-min passive recovery period. The dataset includes participant characteristics, the number of sprints completed during the fatigue protocol, mean and peak power output, blood lactate concentration, mean arterial pressure, vastus lateralis muscle oxygen saturation, total hemoglobin, and rating of perceived exertion. Blood lactate, mean arterial pressure, and perceived exertion were assessed at predefined time points throughout the experimental protocol. Muscle oxygen saturation and total hemoglobin were recorded during each sprint and recovery interval of the repeated-sprint performance test and normalized to the trial-specific baseline value. All participant data have been anonymized.

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Twelve recreationally trained men completed the passive control (CON), placebo compression (PLA), 90 mmHg external pneumatic compression, and 190 mmHg external pneumatic compression conditions in a randomized, single-blind, placebo-controlled crossover design. The four experimental trials were separated by at least 7 days. During the first laboratory visit, participants completed physical assessments and a maximal oxygen uptake test on a cycle ergometer. Cycle-ergometer settings, external sprint resistance, and pneumatic-compression sleeve size were recorded for replication across subsequent visits. During each experimental trial, participants completed a 10-min resting baseline period, a 6-min warm-up, and a repeated-sprint cycling fatigue protocol consisting of repeated 10-s all-out sprints separated by 60 s of passive recovery. The fatigue protocol was terminated when cadence fell below 70 rpm for 5 consecutive seconds or when the rating of perceived exertion reached 17. After the fatigue protocol, participants rested for 3 min before completing the assigned 20-min recovery condition: passive supine rest, placebo compression at 0 mmHg, 90 mmHg external pneumatic compression, or 190 mmHg external pneumatic compression. Immediately after the intervention, post-intervention measurements were obtained. Following an additional 5-min rest and a 4-min warm-up, participants performed five 10-s maximal cycling sprints separated by 60 s of passive recovery. A 30-min passive recovery period followed the performance test. Mean and peak power were recorded using the built-in measurement system of a Monark LC7TT cycle ergometer. Capillary blood lactate was measured using a Lactate Pro 2 analyzer, and mean arterial pressure was obtained from automated brachial blood-pressure measurements. Vastus lateralis muscle oxygen saturation and total hemoglobin were continuously monitored using a Moxy near-infrared spectroscopy device. Rating of perceived exertion was assessed using the Borg 6–20 scale. Statistical analyses were performed using one-way and two-way repeated-measures ANOVA, with Holm–Bonferroni-adjusted pairwise comparisons where appropriate. Additional methodological details are provided in the associated manuscript.

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Exercise Physiology

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