Activity-dependent degradation of Kv4.2 contributes to synaptic plasticity and behavior in Angelman syndrome model mice

Name: Activity-dependent degradation of Kv4.2 contributes to synaptic plasticity and behavior in Angelman syndrome model mice
Creator: Jia-Hua Hu
Published: 2025-04-16T14:44:20.396Z
Keywords: Ubiquitylation, Potassium Channels, Physiology of Long-Term Potentiation, Reversal Learning, Angelman Syndrome

Hu, Jia-Hua; Malloy, Cole; Liu, Ying; Park, Jung; Pratt, Ashley; Welch, Meghyn; Murphy, Jonathan; Abebe, Daniel; Karlsson, Rose-Marie; Cameron, Heather; Hoffman, Dax

doi:10.17632/sk8vnmw2f4.1

Activity-dependent degradation of Kv4.2 contributes to synaptic plasticity and behavior in Angelman syndrome model mice

Published: 16 April 2025| Version 1 | DOI: 10.17632/sk8vnmw2f4.1

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Jia-Hua Hu, Cole Malloy, Ying Liu, Jung Park, Ashley Pratt, Meghyn Welch, Jonathan Murphy, Daniel Abebe, Rose-Marie Karlsson, Heather Cameron, Dax Hoffman

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Raw data for manuscript figures: Hu et al. "Activity-dependent degradation of Kv4.2 contributes to synaptic plasticity and behavior in Angelman syndrome model mice" 2025. Data are organized into individual zip files corresponding to each figure.

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Eunice Kennedy Shriver National Institute of Child Health and Human Development
United States
National Institute of Mental Health
United States

Activity-dependent degradation of Kv4.2 contributes to synaptic plasticity and behavior in Angelman syndrome model mice

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